'The origins of conduct problems' and 'Investigating the neurobiological impact of childhood maltreatment: What can we learn about risk and resilience?'
- Mechanical Engineering basement room B22
- Life and Environmental Sciences, Research
Part of the School Seminar Series
Speaker: Essi Viding
'The origins of conduct problems'
Antisocial behavior is a substantial societal concern and a considerable amount of resources are targeted into its prevention and treatment. However, the success of the prevention and treatment efforts is, at best, moderate. Decades of developmental psychopathology research highlight that we should be mindful of the phenomenon of ‘equifinality’, namely that individuals may present with similar behaviors for a number of different underlying reasons. In this talk I will review studies that have employed a variety of methodologies and focused on heterogeneity among children with conduct problems - i.e. those who present with antisocial behaviour. In particular, I will overview evidence from behavioral, experimental, neuroimaging, and genetically informative studies, which suggest that presence vs. absence of callous-unemotional traits characterize children for whom the origins of conduct conduct problems appear different.
Speaker: Eamon McCrory
'Investigating the neurobiological impact of childhood maltreatment: What can we learn about risk and resilience?'
Childhood maltreatment is associated with later psychopathology, including conduct disorder, anxiety, and depression. However, the neurobiological mechanisms by which childhood adversity heightens vulnerability to psychopathology and the neurobiological processes associated with resilience remain poorly understood. This short presentation will review several recent brain imaging studies reporting structural and functional differences associated with early adversity. It is suggested that these differences are likely to be associated with patterns of psychological adaptation that may for some children increase the risk of later psychopathology.